For Dr. Kathleen Kantak

KKantak

Mashhoon Y, Tsikitas LA, Kantak KM (2009). Dissociable effects of cocaine-seeking behavior following D1 receptor activation and blockade within the caudal and rostral basolateral amygdala in rats. Eur J Neurosci, 29: 1641-1653.

Harvey RC, Dembro KA, Rajagopalan K, Mutebi MM Kantak KM (2009). Effects of self-administered cocaine in adolescent and adult male rats on orbitofrontal cortex-related neurocognitive functioning. Psychopharmacology, 206: 61–71. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2902997/

Wells AM, Janes AC, Liu X, Deschepper CF, Kaufman MJ, Kantak KM (2010). Medial temporal lobe functioning and structure in the Spontaneously Hypertensive Rat: comparison with Wistar-Kyoto Normotensive and Wistar-Kyoto Hypertensive strains. Hippocampus, 20: 787-797. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2878848/

Nic Dhonnchadha BÁ, Szalay JJ, Achat-Mendes C, Platt DM, Otto MW, Spealman RD, Kantak KM (2010).D-cycloserine delays reacquisition of cocaine self-administration by augmenting extinction learning. Neuropsychopharmacology, 35: 357-367. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2928163/

Mashhoon Y, Wells AM, Kantak KM (2010). Interaction of the rostral basolateral amygdala and prelimbic prefrontal cortex in regulating reinstatement of cocaine-seeking behavior. Pharmacol Biochem Behav, 96: 347-353.  http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2940107/

Harvey RC, Sen S, Deaciuc A, Dwoskin LP, Kantak KM (2011). Methylphenidate treatment in adolescent rats with an attention deficit/hyperactivity disorder phenotype: Cocaine addiction vulnerability and dopamine transporter function. Neuropsychopharmacology, 36: 837-847.  http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3055734/

Szalay JJ, Morin ND, Kantak KM (2011) Involvement of the dorsal subiculum and rostral basolateral amygdala in cocaine cue extinction learning in rats. Eur J Neurosci, 33: 1299-1307. http://www.ncbi.nlm.nih.gov/pubmed/21255130

Hofmann SG, Hüweler R, Mackillop J, Kantak KM (2011) Effects of d-cycloserine on craving to alcohol cues in problem drinkers: Preliminary findings. Am J Drug Alcohol Abuse, 38:101-107. http://informahealthcare.com/doi/abs/10.3109/00952990.2011.600396

Nic Dhonnchadha BÁ, Pinard E, Alberati D, Wettstein JG, Spealman RD, Kantak KM. (2012). Inhibiting glycine transporter-1 facilitates cocaine-cue extinction and attenuates reacquisition of cocaine-seeking behavior. Drug Alcohol Depend. 122:119-126.  http://www.ncbi.nlm.nih.gov/pubmed/21992874

Nic Dhonnchadha BÁ, Lovascio B, Shrestha N, Kirkman C, Lin A, Leite-Morris KA, Man HY, Kaplan GB, Kantak KM (2012) Changes in expression of c-Fos protein following cocaine-cue extinction learning. Behav Brain Res, 234:100-106. http://www.sciencedirect.com/science/article/pii/S0166432812004238

Achat-Mendes C, Nic Dhonnchadha BA, Platt DM, Kantak KM, Spealman RD (2012). Glycine transporter- 1 inhibition preceding extinction training inhibits reacquisition of cocaine seeking. Neuropsychopharmacology, 37:2837-2845 http://www.nature.com/npp/journal/v37/n13/full/npp2012155a.html

Szalay JJ, Jordan CJ, Kantak KM (2013). Neural regulation of the time course for cocaine-cue extinction consolidation in rats. Eur J Neurosci, 37: 269-277. https://www.ncbi.nlm.nih.gov/pubmed/23106490

Nic Dhonnchadha BA, Lin A, Leite-Morris KA, Kaplan GB, Man HY, Kantak KM (2013). Alterations in expression and phosphorylation of GluA1 receptors following cocaine-cue extinction learning.  Behav Brain Res, 238: 119-123. http://journals2.scholarsportal.info/details.xqy?uri=/01664328/v238inone_c/119_aieapogrfcel.xml

Kantak KM, Yager, LM, Brisotti MF (2013). Impact of medial orbital cortex and medial subthalamic nucleus inactivation, individually and together, on the maintenance of cocaine self-administration behavior in rats. Behav Brain Res, 238: 1-9. http://www.ncbi.nlm.nih.gov/pubmed/23098798

Harvey RC, Jordan CJ, Tassin DH, Moody KR, Dwoskin LP, Kantak KM (in press). Performance on a strategy set shifting task during adolescence in a genetic model of attention deficit/hyperactivity disorder: Methylphenidate vs. atomoxetine treatments. Behav Brain Res, 244: 38-47. https://www.ncbi.nlm.nih.gov/pubmed/23376704